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Dear Ray We have a Parkinson's website in Malaysia (www.lloydtan-trust.com). Currently we are in the process of making it more complete and informative. I am starting a new section called "What's new in Parkinson's", in which I include the latest research findings, e.g. new medications, stem cell, etc. I have never done this before. So, I'd appreciate if you can advise me on where I can get the materials from, and whether I have to write in to obtain permission. I also wonder whether I can publish some of the articles that you send out in your parkinson's network. I'd appreciate any suggestions from you. Thanks. Dr Chew, Malaysia. > Date: Thu, 9 Jul 2009 14:18:59 -0700 > From: [log in to unmask] > Subject: Interfering with Glutamate to prevent PD > To: [log in to unmask] > > > from Viartis > > 9 July 2009 > > INTERFERING WITH GLUTAMATE TO PREVENT PARKINSON'S DISEASE > > It has been widely reported that researchers are aiming to interfere with > the formation of Glutamate in order to prevent Parkinson's Disease. For the > news reports go to Medical News Today and Science Daily. The research was > recently presented at a conference. Glutamate is able to form GABA in the > brain. GABA is a chemical produced naturally by the brain, that affects > muscular function. An excess of GABA could provoke symptoms of Parkinson's > Disease. The researchers aim to stimulate "trigger points" in order to > prevent the release of glutamate. By targeting specific receptors they hope > that side-effects will be minimised as fewer targets elsewhere in the brain > will be stimulated. They claim that glutamate causes cell death in > Parkinson's Disease. However, glutamate formation is a healthy function, and > has never been shown, in normal quantities, to cause cell death in people > with Parkinson's Disease. The fundamental weakness in their theory is that > glutamate has never been responsible for causing Parkinson's Disease when > dopamine formation is sufficient either. The primary biochemical fault in > Parkinson's Disease has been proven to be the insufficient formation of > dopamine rather than an excess of glutamate. Yet the approach used by the > researchers could not, even in theory, increase dopamine formation. In > order to refer to this article on its own click here. > > Rayilyn Brown > Director AZNPF > Arizona Chapter National Parkinson Foundation > [log in to unmask] > > ---------------------------------------------------------------------- > To sign-off Parkinsn send a message to: mailto:[log in to unmask] > In the body of the message put: signoff parkinsn _________________________________________________________________ More than messages–check out the rest of the Windows Live™. http://www.microsoft.com/windows/windowslive/ ---------------------------------------------------------------------- To sign-off Parkinsn send a message to: mailto:[log in to unmask] In the body of the message put: signoff parkinsn