 |
 |
comments on "clips" of several messages: <<<my question is which proteins are most likely to compete with Carbidopa/Ldopa for the "carriers" in our bloodstream, when we factor in such factors as the digestion times, the amino acids obtained and the recombinations required? If there is any interest, I will tabulate the amino acid content of some of these proteins. Maybe gelatin is good and casein is bad ? Patrick J. Martin..... Might shed some light on which proteins are safest to eat and still maintain a somewhat predictable response to meds. Kathie Tollifson>>> <<<I was wondering that myself, and have been curious as to why some people have experienced reactions from milk. Milk has only 8 grams of protein per 8 oz glass, and 12 grams carbohydrate. Whereas a 3-1/2 oz portion of meat has about 24 grams of protein. I could easily understand meat interfering, but was at a loss to explain the milk. I just received "Good Nutrition in PD" from Susan Reese. I find that the aminos in question appear to be the large neutrals. Much to my surprise, I find that 8 oz of milk contain about half the LNAAs of a portion of lean ham, and about 1/3-1/2 the LNAAs of a serving of beef. So--though the meat serving has about 3 times as much protein as a glass of milk, the milk has proportionately more of the LNAAs. Kathrynne Holden>>> Interesting. I am wondering still if the category "milk" is not specific enough. There is knowledge that fat is digestively in the stomach longest, protein next. How much of the "milk problem of interference" is digestive residence metabolic loss (= digestion) of the levodopa while held in the stomach? In other words, is the major loss in the stomach or in the LNAA absorption from the small intestine? I suppose the levodopa percentage leaving the stomach could be much dependent upon the individual's digestive modus operandi. ron 1936, dz PD 1984 Ronald F. Vetter <[log in to unmask]> http://www1.ridgecrest.ca.us/~rfvetter/