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Barbara Smith cgKen69/16 wrote: << ... His neurologist recently agreed to start him on aricept as she says his dementia is "atypical" and it is worth a try. He is only taking 5mg daily which will be increased to 10mg after six weeks if it helps. Already(after two weeks) many people are noticing the change. He can get out words that he wants to say. He is more interested in the people around him and more responsive to them. He has even begun to read again. he is even better able to communicate his toilet needs. My youngest daughter had a long conversation with him and was in tears with joy. I know that aricept "shouldn't" work with PD, but sometimes "shouldn't" doesn't mean "won't". I am eternally grateful to our doctor for being willing to try new things to help my husband.>> the data seem to be that many are found to have indications of both PD and AD in post mortem examinations. there is probable damage from several types of dementia in those who live longer. the potential also exists that the Aricept migrates to help the AD senile dementia but is not significantly detrimental to the imbalance of acetyl choline in the striatal sites. exceptions prove the rule. new understanding replaces old theory with new when something unexpected becomes disturbing to the researcher. Bradyphrenia is not attributable to the CNS I conjecture. I would take some Aricept to evaluate whether it affected my "slow-thinking" which is often noticeable to me these days. The recall of the word most precise is more often a longer search than used to be the case for me. There is no likelihood that the neurotransmitter chemistry and physical processes are simple in the neural networks. perhaps the next poll will be to obtain the positive or negative resuilts of trying Aricept? -- ron 1936, dz PD 1984 Ridgecrest, California Ronald F. Vetter <[log in to unmask]> http://www.ridgecrest.ca.us/~rfvetter