Yesterday, one of Becky's doctors shared a brief article with her concerning EMF (electromagnetic fields). I have found the source for the article via the PubMed MedLine search engine. The specific search page URL follows. http://www.ncbi.nlm.nih.gov/Entrez/medline.html I used the PubMedID (7744555) as the search criteria with the expectation that I copy the specific article into this message to share it. I have done so below, however, there are a whole mess of related articles, hence this information to allow any who choose to view them. The text from the one message follows: UI - 95263219 AU - Sandyk R TI - A drug naive parkinsonian patient successfully treated with weak electromagnetic fields. LA - Eng MH - Affect MH - Case Report MH - Cognition MH - Dopamine MH - *Electromagnetic Fields MH - Human MH - Male MH - Memory MH - Middle Age MH - Parkinson Disease/*therapy MH - Serotonin MH - Sleep RN - 50-67-9 (Serotonin) RN - 51-61-6 (Dopamine) PT - JOURNAL ARTICLE DA - 19950614 DP - 1994 Nov IS - 0020-7454 TA - Int J Neurosci PG - 99-110 SB - M CY - ENGLAND IP - 1-2 VI - 79 JC - GS4 AA - Author EM - 199508 AB - Brief cerebral application of picotesla (pT) electromagnetic fields (EMF) has been demonstrated an efficacious, revolutionary treatment modality for the therapy of Parkinson's disease (PD) with clinical benefits being evident in all motor aspects of the disease as well as in nonmotor symptoms such as mood, sleep, pain, sexual dysfunction, autonomic regulation and cognitive functions. Since treatment with pT EMF has involved PD patients who were treated with dopaminergic agents at the time they received EMF there may have been a synergistic interaction between dopaminergic drugs and EMF. The present communication concerns a 49-year-old male Parkinsonian patient with stage 3 disability on the Hoehn and Yahr scale (1967) who, in response to brief extracranial applications of pT EMF, demonstrated a marked improvement in motor, depressive symptomatology and cognitive functions and was classified as stage 1 several weeks later. This case is remarkable in that the patient did not receive treatment with dopaminergic drugs prior to or during the course of EMF therapy. It suggests that (a) pT range EMF may be efficacious as a monotherapy for PD and should be considered also as a treatment modality for de novo diagnosed patients, and (b) application of these EMF improves Parkinsonism by a mechanism which involves, among others, augmentation of dopaminergic and serotonergic neurotransmission. AD - NeuroCommunication Research Laboratories, Danbury, CT 06811, USA. PMID- 0007744555 SO - Int J Neurosci 1994 Nov;79(1-2):99-110 Maybe this will help some who wrestle with this designer disease called Parkinsons. Jeff Jones, PT CG for Becky [log in to unmask]